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This regulated necrotic cell death is activated in response to Fas/FasL signaling, CD59 ligation combined with pore formation, mediated by ROS, ceramide, and advanced glycation end products (AGEs) [28]

Since semaglutide further slows gastric emptying as part of its mechanism of action, patients with pre-existing gastroparesis may experience worsening of symptoms including nausea, vomiting, bloating, and abdominal discomfort

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1,48 According to WJWEs efficacy in mitigating DHT-induced follicular miniaturization and oxidative stress in our AGA model, we proposed that it may also benefit to FAGA patients by targeting these pathways

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& Ben-Hayyim G