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Physiological and therapeutic roles of neuropeptide Y on biological functions

GPX4 uses GSH as a cofactor to reduce peroxides (e.g., R-OOH) to the corresponding alcohols (R-OH) and also to reduce free of hydrogen peroxide to water, reducing the accumulation of toxic free radicals (e.g., RO-) [49, 92]

This is reflected in Pompes disease (glycogen storage disease type II), in which deficiency of this enzyme leads to progressive lysosomal glycogen buildup, impaired autophagic flux, and (cardio)myopathy 72

Unquestionably, given the exceedingly high energy demands imposed upon the nigrostriatal system, it seems plausible that aging-associated energy failures might critically contribute to PD-associated neurotoxicity