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Instead of efficiently storing incoming calories as fat, treated adipocytes become more metabolically active, breaking down stored triglycerides and releasing them for energy use

These proinflammatory changes were shown to increase the binding of fluorescently-labeled leukocytes to the GPx1-deficient endothelial cells compared to the wildtype cells [85]

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Here we determined the mechanisms and rate constants for the glutathione/glutaredoxin-dependent redox reactions of the widely used redox probe roGFP2 and addressed the question of how mono- and dithiol glutaredoxins catalyze the reduction of roGFP2(S 2 ) as a model non-glutathione protein disulfide substrate