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Meiser, J., Schuster, A., Pietzke, M., Vande Voorde, J., Athineos, D., Oizel, K

Simulating the TREK-1 receptor may reduce neuron excitability, whereas reducing the receptor activity may increase neuron excitability

Autophagy, by contrast, is best framed as a compensatory quality-control program: although it can rise in parallel with inflammatory stress, its net role is often stabilizing because it removes damaged organelles/protein complexes and limits downstream inflammatory amplification

doi: 10.1530/JOE180074 164 LianALiXJiangQ

It acts as a cofactor along with SHMT for the formation of methylfolate