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Recent advances in the diagnosis of ChurgStrauss syndrome

After lipopolysaccharide (LPS) stimulates the cytoplasm, caspase-4/caspase-5/caspase-11 (the human equivalent of mouse caspase-11 caspase-4/caspase-5) can directly bind to the conserved structure of LPS, lipoprotein A, causing oligomerization, activation, and the N-terminal of GSDMD to be cleaved and localized to the cell membrane to form membrane pores (Kayagaki et al., 2015)

It is critical to understand the etiopathogenesis, altered physiology and impact of the interventions on the pathophysiology in order to manage these patients effectively

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use within 14 days [6]

Conclusion: First evaluation by RRV and further transfer in patient transport ambulance allows adequate identification of acute stroke patients without delaying arrival at the hospital